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However, their dysregulation can exacerbate inflammation and tissue damage in ARDS ( Given the central role of oxidative stress and lipid peroxidation in ARDS, targeting ferroptosis represents a promising therapeutic strategy ( 2 Ferroptosis in infection and ARDS development Cell death pathways play critical roles in immune homeostasis, infection defense, and inflammation resolution

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Together, these observations support a model in which pathological consequences of altered melatonin rhythms in asthma may arise from a temporal mismatch between melatonin availability and NRF2-driven redox programs in key immune cell populations such as ILC2s

Higgins JPT, Green S (editors)