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These conditions of excessive lipid toxicity and metabolic load eventually lead to the accumulation of liver lipids, cell damage, inflammation, and fibrosis (187, 188)

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Similarly, semaglutide inhibits the ubiquitin-proteasome degradation mediated by NF-B/myostatin in the gastrocnemius tissue of diabetic mice with chronic liver disease models, and enhances myogenesis mediated by heat shock factor-1 (HSCF-1)

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Mousavi SM, Mofrad MD, do Nascimento IJB, Milajerdi A, Mokhtari T, Esmaillzadeh A